Lys-(Des-Arg9)-Bradykinin is a bradykinin derivative lacking the C-terminal arginine and extended with N-terminal lysine. Its modified sequence alters receptor subtype selectivity, making it valuable in kinin receptor studies. Researchers use it to dissect structural elements governing receptor discrimination. The peptide aids mechanistic investigations of kinin signaling.
CAT No: B09044
CAS No:71800-36-7
Synonyms/Alias:Lys-[Des-Arg9]Bradykinin;71800-36-7;des-Arg10-Kallidin;kallidin, des-Arg(10)-;LYS-ARG-PRO-PRO-GLY-PHE-SER-PRO-PHE;[des-Arg10]kallidin;(2S)-2-[[(2S)-1-[(2S)-2-[[(2S)-2-[[2-[[(2S)-1-[(2S)-1-[(2S)-2-[[(2S)-2,6-diaminohexanoyl]amino]-5-(diaminomethylideneamino)pentanoyl]pyrrolidine-2-carbonyl]pyrrolidine-2-carbonyl]amino]acetyl]amino]-3-phenylpropanoyl]amino]-3-hydroxypropanoyl]pyrrolidine-2-carbonyl]amino]-3-phenylpropanoic acid;CHEMBL264100;Lys-[des-Arg9]-bradykinin;Bradykinin, lys-desarg(9);10-De-arg-kallidin;Lys-des-arg(9)-BK;Des-arg(10)-kallidin;Kallidin, des-arginine(10)-;Bradykinin, lysyl-des-arginine(9);[Lys-des-Arg9]-Bradykinin;Bradykinin, lys-D-phe(8)-des-arg(9);kallidin1-9;1-9-Kallidin;kallidin[1-9];Lys-[des-Arg9]BK;[Lys,des-Arg9]bradykinin;[3H]Lys-[des-Arg9]BK;GTPL644;[3H]-Lys-[des-Arg9]BK;GTPL3825;SCHEMBL20298769;DTXSID50222058;AILVBOHFGXNHCC-TZPCGENMSA-N;[3H]-Lys-[des-Arg9]-bradykinin;BDBM50156454;BDBM50214792;AKOS024457490;NCGC00167261-01;FL108890;Bradykinin, N2-L-lysyl-9-de-L-arginine-;HY-103295;CS-0027286;[Lys-des-Arg9]-Bradykinin, >=95% (HPLC);Des-Arg10-Kallidin (Lys-des-Arg9 Bradykinin);Lys-(Des-Arg9)-Bradykinin trifluoroacetate salt;Q27077021;H-Lys-Arg-Pro-Pro-Gly-Phe-Ser-Pro-Phe-OH; H-KRPPGFSPF-OH;(2S)-2-{[(2S)-1-[(2S)-2-[(2S)-2-(2-{[(2S)-1-[(2S)-1-[(2S)-5-CARBAMIMIDAMIDO-2-[(2S)-2,6-DIAMINOHEXANAMIDO]PENTANOYL]PYRROLIDINE-2-CARBONYL]PYRROLIDIN-2-YL]FORMAMIDO}ACETAMIDO)-3-PHENYLPROPANAMIDO]-3-HYDROXYPROPANOYL]PYRROLIDIN-2-YL]FORMAMIDO}-3-PHENYLPROPANOIC ACID;
1. High fat diet and GLP-1 drugs induce pancreatic injury in mice
2. TMEM16F and dynamins control expansive plasma membrane reservoirs
3. Adipose tissue is a key organ for the beneficial effects of GLP-2 metabolic function
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